Read on Twitter
Our group decided to compare the susceptibility of S. marcescens to beta-lactam and aminoglycoside antibiotics. Nosocomial infections can be caused by S. marcescens which is why it is important to understand how antibiotics can be used to treat these infections. [1/4]
![Our group decided to compare the susceptibility of S. marcescens to beta-lactam and aminoglycoside antibiotics. Nosocomial infections can be caused by S. marcescens which is why it is important to understand how antibiotics can be used to treat these infections. [1/4]](https://pbs.twimg.com/media/EmfbJXkWMAAZ9Ag.jpg "Our group decided to compare the susceptibility of S. marcescens to beta-lactam and aminoglycoside antibiotics. Nosocomial infections can be caused by S. marcescens which is why it is important to understand how antibiotics can be used to treat these infections. [1/4]")
We used ampicillin (a beta-lactam) and streptomycin (an aminoglycoside ) in our experiment with each treatment having 9 replicated. We hypothesized that the bacteria would be more resistant to ampicillin than streptomycin because S. marcescens are gram negative bacterium [2/4]
![We used ampicillin (a beta-lactam) and streptomycin (an aminoglycoside ) in our experiment with each treatment having 9 replicated. We hypothesized that the bacteria would be more resistant to ampicillin than streptomycin because S. marcescens are gram negative bacterium [2/4]](https://pbs.twimg.com/media/EmfbJuYXUAEs5hc.jpg "We used ampicillin (a beta-lactam) and streptomycin (an aminoglycoside ) in our experiment with each treatment having 9 replicated. We hypothesized that the bacteria would be more resistant to ampicillin than streptomycin because S. marcescens are gram negative bacterium [2/4]")
After statistical analysis of the mean diameter of some of inhibition for each experimental replicate, we found that there was no statistical difference between either treatment. Thus, our finding disagreed with our original hypothesis. [3/4]
![After statistical analysis of the mean diameter of some of inhibition for each experimental replicate, we found that there was no statistical difference between either treatment. Thus, our finding disagreed with our original hypothesis. [3/4]](https://pbs.twimg.com/media/EmfbJ-IXIAAww0T.jpg "After statistical analysis of the mean diameter of some of inhibition for each experimental replicate, we found that there was no statistical difference between either treatment. Thus, our finding disagreed with our original hypothesis. [3/4]")
Our study was only limited to one type of each antibiotic. To address this limitation, we can introduce more beta-lactams and aminoglycosides to our experimental design. This way we can gain a more extensive view on how bacteria develop antibiotic resistance. [4/4]
